Every year we bring you 120+ speakers covering a wide range of topics that touch every area of your family's life. Here is a sample of our exciting speaker roster for 2013. This list is still under construction; many more will follow -- so stay tuned!
Julianne Adams, DSH-P, BCIH, NST-CP
Mark Geier, MD, PhD
Dr. Richard Moskowitz
Jackie de Vries, MS Computer Science, Columbia University; Barbara Brennan School of Healing
Autism disrupts the early childhood developmental process which is the foundation for neurotypical development. Recovery is most complete when paired with therapies that improve brain regulation, enhance the adaptation of physical body systems, release underlying stress and trauma and bridge the developmental gaps. At Crossroads we tailor our multidisciplinary approach for each individual including unique neurofeedback modalities, energy bioregulation systems and integrated somatic therapies. We will share the insights we have garnered on this journey through recovery.
Theresa A Deisher, Ph.D.
Dr. Deisher will summarize 1) the current worldwide autism epidemic through SCPI's new interactive globe of data, 2) the temporal association between the use of human fetal cell lines for vaccine manufacture and autism disorder, 3) introduce the science of auto-immunity and insertional mutagenesis that can be triggered by human fetal manufactured vaccines and 4) discuss clinical and real-life experiments demonstrating the risks of childhood vaccines containing extremely high levels of human fetal DNA and retroviral fragment contaminants.
Richard C. Deth
Methylation of DNA may play a central role in the origin of Down syndrome (non-dysjunction), and the developmental trajectory of Down syndrome individuals, as well as affecting their life-long capabilities and health. Dr. Deth will review our current understanding of neuroepigenetics, particularly as it relates to Down syndrome.
The ability to resist oxidation and to adapt to environmental stressors is fundamental to homeostasis, and sulfur metabolism provides the foundation for these abilities. Signaling molecules with the ability to change the redox state can exert a broad influence over cellular metabolism, including methylation-mediated epigenetic effects on gene transcription that can persist across the lifespan and even across generations. Conversely, environmental factors which interfere with redox signaling can disrupt its regulatory role, contributing to a number of disorders affecting almost every aspect of physiological function. This lecture will provide an overview of the metabolic and biochemical relationships that control the redox state of cells, including tissue-specific differences.
There is extensive evidence indicating the occurrence of oxidative stress, neuroinflammation and impaired methylation in autism and related neurodevelopmental disorders. This knowledge has assisted in identifying treatment approaches which can provide therapeutic benefit for many, but not all individuals. Autism serves to highlight the critical relationship between the GI tract and neurological/immunological development, in which epigenetic regulation plays a key role. The important role of genetic susceptibility can also be viewed within the context of redox and methylation-related metabolism. This lecture will review the scientific evidence for impaired redox and methylation status in autism, including possible causative factors and implications for treatment.
The clinician will:
Learn underlying physiological mechanisms vital to a deeper understanding of autism spectrum disorder
Understand the role of methylation in autism spectrum disorder
Understand the role of oxidative stress and inflammation in autism pathology
Understand the interrelationships of different body systems
Understand epigenetic regulation upon body systems
Brain development is driven by changes in methylation of DNA and histone proteins which combine to provide epigenetic regulation. Methylation depends upon methionine synthase and vitamin B12. B12 status in the brain is distinct from the rest of the body, and brain levels of B12 are markedly lower in autism, schizophrenia and normal aging. Recent studies of the epigenetic effects of gluten and casein-derived opiate peptides indicate their significance for development and life-long disease risk.
Understanding the interrelationship between all of the triggers affecting our children and future generations. The Power of Networking. How to create your own local chapter. Roundtable Discussion & Feedback (Representatives from each roundtable review discussion points, agreements, goals, and objectives). Representatives from each roundtable review discussion points, agreements, goals, and objectives. Outreach Events. Spreading awareness and inspiring families through outreach programs. Roundtable Discussion Roundtable Feedback. The Power of Enrollment. Becoming a one-person agent of change. Top 10 Ways to Make A Difference. Utilizing Media to Get Your Message Out. Uploading photos and videos to FutureForward’s YouTube.com Channel. Posting notices through your local access TV channel; through social media; through phone trees, and others.
Nicholas Dogris, Ph.D, BCN
NeuroField pulsed electromagnetic field (pEMF) stimulation is a new and innovative method that has been designed to address neurological deregulation. In this presentation Dr. Dogris will present an overview of the NeuroField system along with EEG brainwave normative data of several autistic patients. The theoretical and clinical underpinnings of NeuroField will be discussed along with a question and answer period.
Dr. Andreas Ludwig Kalcker , (Ph.D)
Presentation of the Dr. Kalcker's Protocol for healing regressive autism. More than 117 Cured cases in less than 2 years (!) are prove of his hypothesis of Parasitological Vaccination as the true reason for Autism. Toxins released by Parasites are the cause of the symptoms known as regressive autism.
1.Morphine, the gastrointestinal effects of morphine are mediated primarily by μ-opioid receptors in the bowel reducing gut motility
2.MDA (Malondialdehyde), that is a reactive aldehyde and is one of the many reactive electrophile species that cause toxic stress in cells and form covalent protein adducts
3. Amonia, where Hyperammonemia is one of the metabolic derangements that contribute to hepatic encephalopathy.
4. Histamine is causing inflamation and implicated in neurotransmission of peripheral sensory information
5. formaldehyde cause severe injury to the upper gastrointestinal tract.
Applying his special protocol that he developed with Miriam Carrasco and Kerri Rivera together with the correct diet all children improved drastically and complete recovery has been proven to be possible in more cases than ever.
In this presentation Tami will cover the basics of Lyme disease, testing/diagnoses, treatments options and how it plays a role in the entire picture of Autism. In addition she will discuss the things from a mind, body, and spiritual healing perspective which may go unnoticed but be a key element to full healing. Whether your child has Lyme as part of the picture or not this presentation will give you insights into different aspects which may be hindering the healing process. Once these underlying causes are uncovered Tami will give you strategies from a Vibrational Healing perspective to balance these things in your child and your family.
Andi Durkin, BA
Explore the various avenues of support that nutritional, supplemental, medical and environmental changes may offer to a mother with a prenatal diagnosis of Down syndrome. Learn how proper, early intervention— which adds physical, neurodevelopmental, and other research-based strategies—can help optimize the health of the child with Down syndrome.
A method is proposed to enhance brain development before and after birth. Several cases of Fetal Down syndrome are discussed. The literature of Trisomic mouse models of Down syndrome is reviewed.
Scientific research on vaccines has been limited by a narrowness of approach. The current model of government and industry-sponsored research utilizes research methods that sanction the use of active placebos and active controls, which may be masking background levels of adverse reactions. Subjects are followed for short time frames in relation to the biological activity and reactivity of the components, preparations and combinations under review. Passive surveillance is inadequate to assess both short and long term health consequences at the population level due to under reporting and lack of scientific certainty of the relationship between the vaccines and health effects. Government sponsored safety monitoring research is primarily epidemiological. Bench science is required to examine the biological, molecular, genetic, and physiological impacts.
The assumptions that are made about vaccine manufacturing processes, ingredients, and testing help to ensure an outcome that is easy to control or influence. Adjuvants and preservatives have been presumed safe. The risk that culture mediums pose minimal risk of foreign protein and DNA contamination has been ignored, despite the tragic contamination of the polio vaccine with SV40 and the discovery of recent retroviral DNA contamination in some vaccines. In addition, vaccines contain a complex array of untested additives and ingredients that have never been proven safe to inject, nor do we know the cumulative and synergistic impacts of vaccine combinations and multiple exposures. Studies with narrow sets of objectives may be more likely to result in vaccines reaching the marketplace.
Children’s Medical Safety Research Institute and the Dwoskin Family Foundation is funding research into biological and genetic effects after vaccination, and establishing mechanisms of action, with a current focus on aluminum adjuvants and vaccines that contain aluminum. Evidence is mounting that vaccines contain harmful immune and neurotoxins that can cumulatively and synergistically harm the health and development of infants and children. An overview of published research and research in progress will be discussed, as well as ongoing education and outreach efforts of the foundations.
Dr. C. Rick Ellis, Ed.D.
Violence in an ASD family does not appear magically. By understanding the dynamics of stress on the ASD family, appropriate interventions can be provided, thus reducing the probability that aggression will occur. After the fact, it is easy to understand why tragedies sometimes occur. To be proactive, parents need to understand that there is always support and/or appropriate interventions available even when stress appears to reach an "unmanageable" level. A biopsychosocial model of ASD family stress and violence prevention will be presented.