Dr. Boyd Haley The Effects of Synergistic Toxicities and Genetic Susceptibilities

Data now exists that strongly indicates that there is a genetic subset of the human population that is unable to effectively excrete mercury from low level exposures. This leads to a retention toxicity in this subset at levels of Hg exposure that is easily excreted by the bulk of the healthy population. Autistic children seem to fit into this subset. The observed low levels of mercury in the blood, urine and hair of autistics, when compared to the higher levels retained in their other body tissues, indicates that retention toxicity occurs. This susceptible subset of the population, due to the low frequency, is very likely to be overlooked or not be apparent in most epidemiological studies that consider general populations. However, individuals with the inability to excrete mercury would be expected to develop neurological problems such as autism, AD, etc. Sorting out these individuals and comparing their toxicity status to the general, healthy population produces results strongly indicating that mercury exposures may be the cause of their neurological problems. Recent data also implicate mercury and mercury containing compounds as the likely cause of autism by an environmental toxin based on the fact that about 4 of 5 autistics are male and mercury is well known in the scientific literature to be lethal to male test animals more than females. Data is presented showing the effect is likely due to effects of estrogen versus testosterone on mercury retention.
 
An evaluation of the relative toxic effects of mercury and thimerosal show that the younger the infant the more toxic and lethal the effects can be. Mercury is primarily excreted in the feces of healthy subjects via the billary transport system of the liver, which is not developed in the youngest infants. An evaluation of the considerations of synergistic toxicities, genetic susceptibility, gender and infant age will be presented along with the basic biochemical and cellular level research that strongly supports the thimerosal causation of autism hypothesis. It is now well known that mercury in the fetal cord blood is much higher than in the mother's blood indicating that the fetus is being exposed to larger amounts of mercury per unit body weight than is the mother.
 
The retention and toxicity of mercury and mercury compounds is known to be enhanced by the presence of other synergistic factors that may or may not have toxicity by themselves. Such non-toxic compounds include antibiotics, a milk diet and male hormone. Toxic compounds such as other heavy metals (lead, aluminum) are well known to dramatically increase the toxicity of low levels of mercury exposures. Therefore, unless a total knowledge of the exposure to synergistic toxins are know it is impossible to define a safe level of mercury exposure for humans in the environment.
 
Boyd Haley, PhD served as a medic in the U.S. Army and obtained his PhD in chemistry/biochemistry at Washington State University. He was an NIH Postdoctoral Scholar in the Department of Physiology, Yale University Medical School. His first academic appointment was at the University of Wyoming in 1974 where he was promoted to full professor in 1983. In 1985 he was hired by the University of Kentucky Markey Cancer Center with academic appointments as professor in the College of Pharmacy in the Division of Medicinal Chemistry and in the Department of Biochemistry. He was appointed to be chair and professor of chemistry/biochemistry in the Department of Chemistry from 1996 to 2005. He retired from the University of Kentucky in July 2008. He has lectured throughout the world and testified before congressional committees and the Institute of Medicine regarding various aspects of mercury toxicity and neurological diseases.
 
 
Presentation includes mercury depletion of molybdenum which leads to the inhibition of sulfite conversion to sulfate which leads to lower mylenization of the central nervous system in autism and other neurological illnesses; oxidative stress as a common occurrence in autism including aspects concerning causation and dietary treatment; and low glutathione levels in people of all ages as a major risk factor for many viral infections including influenza.

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