A Deficiency in the Mitochondrial Enzyme, MAO-A, May Be a Possible Etiology of Autism by Elaine DeLack, RN

On September 21, 2010, 6:36 pm

This presentation will explain how the metabolic imbalances, gastric problems, and core symptoms of autism may be the result of a malfunction of the mitochondrial enzyme, MAO-A. The presentation will discuss how stress, genetics, epidurals with Pitocin (oxytocin) augmentation during childbirth, toxins such as mercury, aluminum, high copper and cadmium are possible risk factors contributing to the overall symptoms resulting from the MAO-A deficiency. This science provides the rationale unifying the benefits seen from various therapies such as the gluten-free and casein-free diet, hyperbaric oxygen, methylcobalamin and Respen-A.

Elaine DeLack, RN is a board certified registered nurse whose work focuses on research and treatment options in multiple sclerosis, depression, autism and Alzheimer’s. Her scientific publications have appeared in the journals Multiple Sclerosis Clinical and Laboratory Research and Alternative Medicine Review. She is also the author of the book They Said It Didn’t Make ‘Cents’ – MS the Prokarin Story, in which she tells her life story and the struggles she has encountered in trying to bring Prokarin into mainstream medicine as a treatment option for MS. Formerly she worked in the care of the elderly and those with chronic disease in transitional care facilities. In the past 17 years she has been active in the advancement of new treatments for multiple sclerosis and related disease states. She has developed and patented treatments for MS, depression and bipolar depressive disorder, and ASD. She has lectured throughout the US and Canada to patients, doctors and pharmacists on issues of medical research and study application. She is the CEO and founder of two medical research companies.

Your rating: None Average: 5 (1 vote)